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Molecular Cardiology Research Institute
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Principal Investigators









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Debbie Beasley, Ph.D.

  Associate Professor of Medicine
Laboratory of Vascular Myocyte Biology

Tufts Medical Center

800 Washington Street, Box 8486

Boston, MA 02111

   617-636-8248

Debbie Beasley    

 

 Biosketch




Dr. Beasley received her doctoral degree in Physiology from the University of Michigan. She completed postdoctoral training at Boston University Medical Center, and Brigham and Women’s Hospital, and was appointed Assistant Professor of Medicine at Harvard Medical School in 1991. Currently, she is the director of the Laboratory of Vascular Myocyte Biology, and is an Associate Research Professor of Medicine at Tufts University School of Medicine.

 

Research Administrator: Dionne Marshall

Schultz K, Murthy V, Tatro JB, Beasley D.  Endogenous Interleukin -1 promotes a proliferative and proinflammatory phenotype in human vascular smooth muscle cells.  American Journal of Physiology - Heart and Circulatory Physiology 2007;292(6):H2927-H2934.  PubMed Abstract

 

Schultz K, Fanburg, BL and Beasley D.  Hypoxia and hypoxia-inducible factor-1 promote growth factor-induced proliferation in human vascular smooth muscle cells.  American Journal of Physiology - Heart and Circulatory Physiology 2006;290(6):H2528-H2534.  PubMed Abstract

 

Yang X, Murthy V, Schultz K, Tatro JB, Fitzgerald KA, Beasley D.  Toll-like receptor 3 signaling evokes a proinflammatory and proliferative phenotype in human vascular smooth muscle cells.  American Journal of Physiology - Heart and Circulatory Physiology 2006;291(5):H2334-H2343.  PubMed Abstract

 

Yang X, Coriolan D, Schultz K, Golenbock DT, Beasley D.  Toll-like receptor 2 mediates persistent chemokine release by chlamydia pneumoniae-infected vascular smooth muscle cells.  Arteriosclerosis Thrombosis, and Vascular Biology 2005;25(11):2308-2314.  PubMed Abstract

 

Yang X, Coriolan D, Murthy V, Schultz K, Golenbock DT, Beasley D.  Proinflammatory phenotype of vascular smooth muscle cells: Role of efficient toll-like receptor 4 signaling.  American Journal of Physiology - Heart and Circulatory Physiology 205;289(3):H1068H1076.  PubMed Abstract